Blood Vessels: Problems Arising at the Borders of Natural by F. Hammersen (auth.), Professor Dr. med. Sven Effert,

By F. Hammersen (auth.), Professor Dr. med. Sven Effert, Priv.-Doz. Dr. med. Johannes Dietrich Meyer-Erkelenz (eds.)

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If the erythrocytes are made adhesive they stick to the wall in the stagnation region where they aggregate and form leaflets of irregular thickness (2 ~ 20 ~m) covering a defined area of the wall surface. These erythrocyte depositions can only be removed by increasing the flow velocity at the wall within the dead water. The distribution of the hydrodynamic force components at the wall within the dead water region can approximately be derived from the velocity-field (lower part of Fig. 1). In the representation of velocity components the stagnation point is characterized by a'maximum velocity component towards the wall and perpendicular to it (vl solid line in the diagram, upper part of Fig.

The addition and removal of DMSO and the freezing program were modified after the methods of KIM and BALDINI (1973) and MURPHY et al. (1974). As indicated in the figures there is a lower recovery and a shorter survival of the frozen platelets. The damage that has been caused by 52 % Recovery • = Radioakt. 0= Pliiftchen-Zahl Rec = 34% ~xp=2,3d 2 3 4 5 6 Tage post transf. Fig. 1. (a) Recovery and survival of transfused f~esh human platelet concentrates. For explanation of symbols see text % Recovery • = Radioakt.

Furthermore, it is generally accepted that by contributing to a gradual thickening of the intima, mural thrombi are a frequent early lesion in the development of atherosclerotic plaques (MOVAT, HAUST and MORE, 1959). It is also known that stenosing atherosclerosis of muscular arteries does not occur without preceding nural thrombosis. The increasing incidence of occlusive arterial disease in most western countries has made the determination of the role of platelets in arterial thrombosis a problem of major importance.

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